American Philosophical Society
Member History

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Residency
Resident (1)
Class
1Name:  Dr. Carol W. Greider
 Institution:  Johns Hopkins University School of Medicine
 Year Elected:  2016
 Class:  2. Biological Sciences
 Subdivision:  201. Molecular Biology and Biochemistry
 Residency:  Resident
 Living? :   Living
 Birth Date:  1961
   
 
Carol Greider, Ph.D. received her bachelor’s degree from the University of California at Santa Barbara in 1983 and a Ph.D. in 1987 from the University of California at Berkeley. In 1984, working together with Dr. Elizabeth Blackburn, she discovered telomerase, an enzyme that maintains telomeres, or chromosome ends. In 1988, Dr. Greider was recruited to Cold Spring Harbor Laboratory as an independent Cold Spring Harbor Fellow, where she cloned and characterized the RNA component of telomerase. In 1990, Dr. Greider was appointed as an assistant investigator at Cold Spring Harbor Laboratory, followed later by appointment to Investigator in 1994. She expanded the focus of her telomere research to include the role of short telomeres in cellular senescence, cell death and in cancer. In 1997, Dr. Greider moved her laboratory to the Department of Molecular Biology and Genetics at Johns Hopkins University School of Medicine. In 2003, she was appointed as the Daniel Nathans Professor and Director of the Department of Molecular Biology and Genetics. Dr. Greider’s group continued to study the biochemistry of telomerase and determined the secondary structure of the human telomerase RNA. In addition, she characterized the loss of telomere function in mice, which allowed an understanding of human diseases that make up the short telomere syndromes. Dr. Greider shared the Nobel Prize in Physiology or Medicine in 2009 with Drs. Elizabeth Blackburn and Jack Szostak for their work on telomeres and telomerase. In 2014, Dr. Greider was appointed as a Blooomberg Distinguished Professor at Johns Hopkins University. Dr. Greider currently directs a group of eight scientists studying both the role of short telomeres in age-related disease and cancer as well as the regulatory mechanism that maintain telomere length.
 
Election Year
2016 (1)